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High - Renin HTN

Normal - Renin HTN

Low - Renin HTN

High Renin Normal Renin Low Renin

Renin HTN

Low-Renin Hypertension accounts for 20-30% of Hypertensive Patients

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In these conditions, the cause of aldosteronism is made by recognizing the underlying disease. The Differential Diagnosis of Secondary Hyperaldosteronism is sorted out by the presence or absence of HTN and the presence of other obvious diagnoses (CHF/hepatic dysfunction, dehydration, pregnancy, etc).  Source: Halperin F., Alexander E., Robert Dluhy R.  Endotext: Chapter 23 – Aldosterone Excess July 15 2010.Note:  dopamine, heparin and ANP (Atrial Natriuretic Peptide) inhibit the production of Aldosterone. Potassium and ACTH stimulate Aldosterone production.

Secondary aldosteronism is more common than primary aldosteronism. It is caused by anything that leads to excess aldosterone, other than a disorder of the adrenal glands. It could be caused by anything that decreases blood flow to the kidneys, decreases blood pressure, or lowers sodium levels, i.e. anything that increases the activation of the renin-angiotensin system. Secondary aldosteronism may be seen with congestive heart failure, cirrhosis of the liver, kidney disease, and toxemia of pregnancy (preeclampsia). It is also common in dehydration

High - Renin HTN